- SUPPORTS NERVOUS SYSTEM: B6/B1 Plus Zinc contains active forms of vitamins B6 and B1, both recognized for their importance in brain function and the nervous system.
- AIDS IN COGNITION AND BRAIN STRUCTURE: Vitamins B1 and B6 work together in a complex interplay and play key roles in cognition, brain structure and functional connectivity in older adults.
- HEALTHY METABOLISM: B6/B1 Plus Zinc contains 5 mg of highly bioavailable zinc, acting as a cofactor in over 300 enzymatic reactions, supporting a healthy metabolism.
An increased level of catabolism of vitamin B6 has been observed during inflammation or activated cellular immunity, resulting in a decreased circulating level of vitamin B6.
Vitamin B6 is required by enzymes called transaminases in the first step of the breakdown of most amino acids for energy production. Sometimes consuming a meat-focused diet can require more B6.
Vitamin B6 and B1 are especially important for proper function of nervous system function, and over 300 different enzymes are dependent on zinc to function properly, including the ability to make hydrochloric acid.
When we are deficient in B6, some pathways break down, such as the ability to turn hydroxyproline (collagen) into glycine. With the B6 deficiency, more of the hydroxyproline will be shuttled to make oxalic acid (oxalates). B6 can also contribute to excess yeast growth, such as candida.
One way to check if this is properly functioning, is by taking the Organic Acids Test.
Vitamin B1 (thiamin) is most strongly linked with carbohydrate metabolism, but it is also required for the oxidative decarboxylation of other alpha-keto acids derived from certain amino acids.
Vitamin B1 is also noted for its benefits for inflammation of the nerves (neuritis), and deficiency is associated with “high calorie malnutrition.”
Zinc serves as an essential cofactor for greater than seventy (70) enzymes, and participates in binding DNA as a component of the zinc binding proteins, which consists of a combination of the protein and zinc. Zinc also functions in many other roles in the body, including participation in the role of insulin release.
Zinc functions in membrane stabilization, and is part of the bone mineral apatite. Zinc has also been demonstrated to “increase osteogenic effect” via multiple mechanisms, increased synthesis of collagen in osteoblastic cells, and via a concentration increase of cellular collagen.
A deficiency in zinc results in a decrease in bone weight, delays growth in bone metabolism, and retards bone growth, development and maintenance. Both in vivo and in vitro studies have also demonstrated that zinc possess stimulatory effects on bone formation and mineralization.
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